Atherosclerosis Treatment
Recovery For
Atherosclerosis Treatment
- Plant Sterols Known To Reduce Cholesterol
- Supports The Cardiovascular System
- Assists The Body To Rebuild Arterial Tissue
- Potent Antioxidants That Benefits The Heart
- Prevents Degeneration
- Award Winning Natural Product
Benefits Of Recovery™ For Atherosclerosis:
- Decreases free radical activity which is a contributing factor linked to atherosclerosis.
- Brings more hydration to the arterial cell and increases cell function and energy.
- Increases arterial cell membrane receptivity, sensitizing cell repair, maintenance, protection and communication.
- Recovery™ is an all-natural food-based non-drug supplement proposed to improve the quality of life and provide excellent alternative atherosclerosis relief.
- May be safely combined with other atherosclerosis medications or taken on its own to help improve the quality of life.
- Since Recovery™ works to modify your body's responses; it may take up to six weeks for you to experience the benefits for your atherosclerosis condition, with most people noticing benefits within a month.
Recovery™ Contains Nutricol®
- A highly concentrated proprietary bioflavonoid formula comprised of theaflavin, EGCG, resveratrol and proanthocyanidins, which are derived from grape and tea.
- These anti-catabolic ingredients will stop the progress of arterial inflammation, free radical activity and tissue damage.
- All Natural Nutricol® decreases trauma from chronic arterial damage, surgery, injury and over training.
- Increases the cell's resistance to damage and improves its ability to repair damage to the arterial area.
Nutricol™ is proposed to increase the structural integrity of arterial connective tissue structures and to maintain optimum tissue hydration. The potential results after using Recovery™ are:
- Decreased susceptibility to injury
- Decreased tissue breakdown
- Faster recovery rates
- Decreased lactic acid buildup
- Decreased pain and inflammation
- Reinforcing membrane and matrix structures through increasing aldimine reducible cross-linking of collagen fibers. This reinforces the strength and elasticity of connectivetissue structures such as fascia, dermis and blood vessel walls.
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ATHEROSCLEROSIS - Information
- What is atherosclerosis?
- How common is atherosclerosis?
- Atherosclerosis and artery inflammation
- What are atherosclerosis warning signs and symptoms?
- Atherosclerosis risk factors
- Atherosclerosis wellness tips
- Recovery with Nutricol - Mechanism of action
- Safety of Recovery
- Safely combining with drugs
- Safety data
- Effect on liver function
What Is Atherosclerosis?
Atherosclerosis is a disease where a fatty, wax-like substance “plaque” is deposited in the arterial walls causing the arteries to constrict and narrow. Eventually, the narrowing of the arteries prevents the blood from flowing appropriately, resulting in heart attack, congestive heart failure or stroke.
Atherosclerosis plaque is comprised of cholesterol, fatty materials, calcium, and cellular waste products. If a section of this plaque breaks off, it can travel into the bloodstream to the point where the plaque can completely block the narrowed artery. If the affected artery supplies the brain, a stroke may result, and if it supplies the heart, a heart attack may result.
Atherosclerosis can also cause high blood pressure “hypertension”. Since the blood is forced through the constricted arteries, it will exert further pressure on the walls. This inevitably increases the risk of heart attack and stroke. Angina “chest pain” is another condition, which is caused by the narrowing of the arteries “atherosclerosis”. At this point atherosclerosis symptoms may occur or atherosclerosis disease may be identified. Once identified, atherosclerosis treatment must be sought.
How Does Atherosclerosis And Atherosclerosis Disease Progress?
Atherosclerosis and atherosclerosis disease is a slow, complex condition that begins in childhood. It is expected to get worse as more plaque is deposited within the arteries. In some individuals, even those in there 30s, atherosclerosis disease can progress quickly.
While the exact cause of atherosclerosis disease remains unidentified, numerous researchers believe that the build up of plaque begins when the deepest layer “intima or endothelium” of the artery becomes damaged by attack of free radicals (oxidation) and coupled by inflammation. When the body attempts to repair this damage, fatty material (such as vLDL cholesterol and LDL) and minerals (such as calcium or other metals) accumulate at the damaged site. This plaque build up constricts the affected artery opening causing atherosclerosis symptoms or atherosclerosis disease.
Note: Arteries deliver blood rich in nutrients and oxygen to the tissues in the body, providing the fuel necessary for the cells to function accurately and repair efficiently. When the arteries narrow, as in the case with atherosclerosis, less oxygen and nutrients will be delivered to the tissues. An inferior nutrient and oxygen supply will lead to loss of tissue function and progressive cell degeneration and death. It is imperative that atherosclerosis treatment is initiated in order to stop the progression of this disease.
How Common Is Atherosclerosis?
It is hard to determine what percentage of the population has atherosclerosis or atherosclerosis disease. Although, approximately one in six North Americans has some form of cardiovascular disease. Most of these diseases usually begin with atherosclerosis.
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Atherosclerosis And Artery Inflammation
Recently, numerous heart experts have been examining the role inflammation plays in the development of atherosclerosis symptoms, atherosclerosis, and atherosclerosis disease, as well as other advanced stages of heart disease. Experts now believe that there is a connection between the inflammatory process, arterial damage, and coronary atherosclerosis.
One of the first indications of inflammation within the arteries is the presence of a substance called (CRP) "C-reactive protein" in the blood. The levels of this indicator (C-reactive protein) are eminent when heart disease is present since the plaque in diseased arteries is typically comprised of inflammatory cells. In a study of 388 males aged 50-69, the majority of coronary artery disease increased 1.5 folds for each doubling of the CRP levels.
It suggests that the reduction of inflammation and damage inside the arteries may hinder plaque build up and thus reducing the risk of atherosclerosis, atherosclerosis symptoms and disease, as well as associated heart conditions.
What Are Atherosclerosis Warning Signs And Atherosclerosis Symptoms?
Specific atherosclerosis symptoms of atherosclerosis or atherosclerosis disease depend on what arteries are affected.
When arteries to the brain are affected, atherosclerosis symptoms and warning signs include:
- Dizzy spells
- Headaches
- Memory problems
- Ringing of ear
- Mood changes
- Poor concentration
When arteries to the heart are affected, atherosclerosis symptoms and warning signs include:
- Elevated blood pressure
- Chest pain (angina)
When the arteries to the arms or legs are affected, atherosclerosis warning signs and symptoms include:
- Exhaustion / fatigue
- Aching muscles
- Pain in the thighs and hips, which may be apparent depending on which arteries may be blocked.
- Cramping in the calves (intermittent claudication)
What laboratory tests assess heart attack risk?
A wide range of markers are used to assess heart attack including:
- CRP (c-reactive protein)
- Homocysteine
- Transferrin
- Total cholesterol, HDL and LDL
- Fibrinogen (a globulin that affects blood coagulation)
- ApolipoproteinB (apoB) and apolipoproteinA-1 (apoA-1) levels, as well as apo ratios - the higher ApoB/Apo A-1 ratio may signal an increased risk of cardiovascular disease. (apoB carries LDL and VLDL cholesterol) (apoA-1 carries HDL cholesterol)
One conclusive test that can evaluate these significant heart health predictors from one blood sample is referred to as the Comprehensive Cardiovascular Report (CCR), which is available from Great Smokies Diagnostic Laboratories.
If you have concerns, talk to your physician and request the CCR test. There are other similar tests available as well. This will provide a general assessment of your heart health risks. Simply writing a prescription for statins to manage your individual symptoms is an unsatisfactory method of treatment.
Scientific opposition to the cholesterol hypothesis
Statin Drugs - A Critical Review of the Risk vs Benefit Research
*Mendall MA, Patel P., Ballam L., et al. C-reactive protein and its relation to cardiovascular risk factor: A population based cross sectional study. BMJ. 1996;312:1061-1065.
Atherosclerosis Risk Factors
Because atherosclerosis accompanies or precedes numerous circulatory and cardiovascular conditions such as high blood pressure, stroke, angina, congestive heart failure, and heart attack, the risk factors associated with these diseases tends to be comparable to symptoms of atherosclerosis. Risk factors for atherosclerosis include:
Factors you can influence:
- High cholesterol levels, especially bad cholesterol such as LDL.
- High levels of blood triglyceride.
- Being a smoker or being exposed to cigarette smoke.
- Elevated blood pressure levels.
- Being diabetic.
- Diet. Consuming fatty foods containing rich hydrogenated oils including over processed foods are thought to contribute to heart disease.
- Living an inactive lifestyle.
- Being obese or overweight.
- Elevated C-reactive protein (CRP) levels in the blood.
- Excessive stress.
Factors you can’t influence:
- Family history of stroke or heart attack before the age of 65 or having the predisposition to develop cholesterol or high blood pressure.
- Your age: being over 45 for men and over 55 for women.
- Ethnicity: African, First Nation, and South Asian ethnicity are at higher risk.
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Atherosclerosis Wellness Tips
Although certain risk factors cannot be primarily addressed for heart disease (family history, your ethnicity and age) many underlying factors are directly influenced by the lifestyle you lead. Here are some simple modifications you can make to help decrease your chances of developing atherosclerosis or other heart related disease.
- If you smoke, consider quitting. Many effective products can be purchased from your pharmacy to help reduce your cravings. The Internet can also be a valuable resource for information on what “quit smoking programs” are available.
- Being overweight. Contact your physician about initiating a weight- reduction plan. Consuming a sensible diet, including a standard exercise program will help you loose any additional weight.
- Start exercising. Research shows that moderate exercise such as a half an hour walk, six days a week, can decrease triglyceride levels, keep cholesterol levels balanced, and can help in weight reduction. A regular exercise program can also assist in stress management and can keep your lungs, heart and muscles working more efficiently..
- Clean up your diet. Consume high-lignan flax oil, hemp oil, or pumpkin oil and include more fatty fish like sardines and salmon into your diet. All of these contain omega 3 fatty acids, which are health-promoting foods. Studies have revealed that diets that include omega-3 fatty acids can lower triglyceride levels, decrease platelet clumping or aggregation, and will also improve the function of the cells which line the heart and the blood vessels. Include fresh fruit, vegetables (preferably organic) and legumes (beans, peas) into your diet. Eat turmeric, ginger and garlic; they all have essential benefits on the heart.
- Don’t consume "bad fats". Try eliminating hydrogenated oils “trans-fatty acids”, from your diet, as they encourage heart disease. Eat fewer foods that are rich with animal fats like beef and pork, including dairy goods that are high in fat.
- Eat smaller, more frequent meals and integrate quality protein foods throughout the day. This will help increase insulin sensitivity, which is essential for individuals who have diabetes. If excess blood sugar “glucose” levels are not kept under control it can create damage to the arterial lining “glycosylation”. Therefore, it is important to regulate your blood sugar levels.
- Take coenzyme Q10 (CoQ10) supplements. CoQ10 is an essential antioxidant, which the heart utilizes for protection against the damaging compounds by the cells during the metabolic process. The heart’s production of CoQ10 diminishes as we age. Drugs such as HMG CoA reductase inhibitors (cholesterol lowering drugs) can also reduce heart protecting CoQ10 levels.
- Take adequate amounts of calcium. Reducing fat-rich dairy foods from your diet can have a direct impact on your calcium intake. Calcium is necessary for normal function of the cardiovascular system. Recent studies have revealed that individuals who have adequate calcium in their diet have a reduced risk of developing high blood pressure, which is directly linked to stroke and heart disease. Consider taking a calcium supplement if you are not getting adequate amounts in your diet. To help with absorption and maintain a calcium/magnesium balance, take calcium supplements that also contain vitamin D and magnesium. 1,200 mg a day is the recommended daily dosage for adult women, between 1,500 and 1,800 mg a day if you are pregnant and/or lactating, 1,500 mg a day if have experienced menopause. Men 19 years or older should take 1,000 mg of calcium per day.
- Take adequate amounts of magnesium. Not eating sufficient green vegetables and grains can affect your magnesium levels. Magnesium is essential for the cardiovascular system to function normally. This mineral is important for the smooth muscles, which surround the blood vessels relax. Recent studies have revealed that individuals who have adequate magnesium in their diet have a reduced risk of developing high blood pressure, which is directly linked to stroke and heart disease. Consider taking a magnesium supplement if you are not getting adequate amounts in your diet.
- Take adequate amounts of B vitamins. The B vitamins in folic acid, B6 and in particular B12 help decrease homocysteine levels within the blood. High homocysteine levels in the blood can increase the development of arteriosclerosis. Fruits, green, leafy vegetables and other natural foods are excellent sources of B vitamins. You may want to include a multi-vitamin to ensure that you are consuming adequate levels of B vitamins daily. Citrus fruits, leafy green vegetables, and pinto, tomatoes, and navy or kidney beans are all rich sources of folic acid “folate”.
- Manage stress effectively. Excessive or mismanaged stress levels play a significant role in elevated blood pressure and heart disease. Educate yourself or consider taking a stress management course on how to manage stress effectively. For general information on reducing stress, see Developing a Positive Attitude under General Wellness Tips.
- Seek advice from your physician or healthcare provider about new food-based non-drug products, which are formulated to improve the quality of life. Recovery with Nutricol™ has been developed to improve circulation to the tissue, speed the repair of tissue, and reduce or halt tissue damage.
- Investigate new options such as Chelation therapy to improve circulation and unblock the arterial walls. Extensive data has established the effectiveness of chelation therapy contrary to varying opinions expressed thoughout the medical profession. Chelation therapy improves the overall health of your arteries.
- If you have been diagnosed to have excessive iron blood levels as a result of hemochromatosis, seek treatment for this condition. Meet with your physician to discuss ways to stabilize your iron levels so that unnecessary damage is not done to blood vessels.
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Recovery™ With Nutricol® - Mechanism Of Action
The significant fat and water soluble antioxidant properties of Nutricol® produce these anti-inflammatory and anti-catabolic effects:
1. Stabilizes collagen aldimine reducible cross-links to reinforce the elasticity and strength of the connective tissues such as with tendons, ligaments, cartilage, synovium, fascia, blood vessel walls, bone and the dermis of the skin.
2. Neutralizes ROS and catholic enzymes, which decrease the negative impact on cellular and extra-cellular functions and structure. This improves membrane’s receptiveness to growth factors such as thyroxin, somatomedins and insulin, which are necessary for cell protection and anabolic repair.
3. Decreases excessive production of catabolic materials such as elastase, collagenase, hyaluronidase, NOS, TNF, and xanthine oxidase. These substances are released from microbial, immune, and damaged cells; therefore causing damage to epithelial and connective tissue. This results in capillary fragility, joint pain, inflammation and further soft-tissue damage.
4. Prevents the release of inflammation creators such as serine proteases, histamine, leukotrienes and prostaglandins by non-competitively obstructing further release of the pro-inflammatory enzymes cyclo-oxygenase, phosphodiesterase and lipoxygenase.
5. Improves the protective epithelial mucosal surface integrity (respiratory, digestive, and genitourinary tract).
Note: When the body utilizes oxygen, it produces an extremely reactive “exhaust” that can be damaging to the cells. The reactive molecules in this “exhaust” are referred to free radicals or reactive oxygen species (ROS). Although certain free radicals play an essential part of the metabolic process, an excessive quantity of them is considered by numerous researchers to be the primary cause of inflammation, cell destruction, aging and degenerative diseases. The diseases, which attribute to augmented free radical action are arthritis, diabetes, cataracts, digestive diseases, breathing conditions, liver disease, heart disease, macular degeneration (age associated vision loss), auto immune diseases and cancer.
The bioactive components of Nutricol® have been revealed in numerous studies to control the production of free radicals in both humans and animals. It is proposed that regular use of this product will help decrease the rate of cell damage associated with free radical actions.Safety Of Recovery™
Recovery™ has significant benefits with very low risk. All Recovery™ ingredients are naturally-occuring and non-toxic.
Nutricol® constituents have been clinically observed to possess anticarinogenic properties in the liver, lung, breast, pancreas, bladder, prostate, skin and most of the gastrointestinal system.
Click here to see what health professionals are saying about Recovery™ with Nutricol™.
Safely Combining With Drugs
Due to Recovery’s antioxidant, anti-catabolic and anti-inflammatory action, combing Recovery™ with prescription drugs can help reduced drug toxicity and side effects:
Anti-inflammatory (NSAIDS/cox-2 inhibitors)
The most commonly prescribed NSAIDS interfere with prostaglandins and cyclo-oxygenase. Cell damage will still continue because:
1. Oxidation of the membranes remain unblocked.
2. With regular NSAIDS, production of PG1 and PG3 are blocked as well. These are typically included in the repair process.
Recovery™ benefits alone or combined with NSAIDS include:
1. Prevents the inflammatory flow or “domino effect” by increasing the cell’s capacity to neutralize lysosomal enzymes and ROS, which are released from nearby damaged cells, therefore reducing further trauma.
2. Increases the delivery of particular neurochemicals, hormones and nutrients into the cells, and enhances the transfer of waste out of the cells. This improves overall cell communication.
Research has revealed that the introduction of the ingredients in Recovery™ with Sulindac (NSAIDS) results in a reduction in GI side effects that accompany Sulindac usage and a synergistic effect in prevention of colon cancer in rats.
Safety Data
Recovery™ has significant health benefits with minimal risks. All Recovery™ ingredients are natural occurring and non-toxic.
Nutricol™ constituents have been clinically recognized to have anticarcinogenic properties in the lung, liver, pancreas, prostate, bladder, breast, skin and the majority of the gastrointestinal system.
Effect on Liver Function
Due to the anti-catabolic and anti-oxidative actions, Recovery™ can assist in the appropriate metabolism and elimination of drugs and other toxins by supporting 4 phase II liver pathways (taurine conjugation, glutathione conjugation, methylation, and sulfation).
